The mechanism of action of Vidalista 5 mg is based on its active ingredient, Tadalafil, which belongs to a class of medications called phosphodiesterase type 5 (PDE5) inhibitors. These drugs primarily work by inhibiting the enzyme phosphodiesterase type 5, which is responsible for the degradation of cyclic guanosine monophosphate (cGMP) in the smooth muscle cells of the penis.
Here’s a simplified explanation of how Vidalista 5 mg works:
Sexual Stimulation: Sexual arousal triggers the release of nitric oxide (NO) in the erectile tissue of the penis.
Increased cGMP Production: Nitric oxide stimulates the production of cyclic guanosine monophosphate (cGMP) from guanosine triphosphate (GTP) in the smooth muscle cells of the penis.
Relaxation of Smooth Muscles: Elevated levels of cGMP cause the smooth muscle cells in the penile arteries to relax, allowing increased blood flow into the penis.
Erection Formation: The increased blood flow into the penile tissues leads to engorgement of the penis, resulting in an erection.
Prolonged Erection: By inhibiting the enzyme phosphodiesterase type 5, Tadalafil prevents the degradation of cGMP, thereby prolonging its effects on smooth muscle relaxation and maintaining the erection.
It’s important to note that Vidalista 5mg, like other PDE5 inhibitors, requires sexual stimulation to be effective. It does not cause spontaneous erections but rather enhances the natural physiological response to sexual arousal.
For more detailed information on the mechanism of action and pharmacology of Vidalista 5 mg, you may refer to medical literature, pharmacology textbooks, or prescribing information provided by the manufacturer. Additionally, reputable medical websites and databases may also provide comprehensive information on the drug’s mechanism of action.
Call: 876-927-9941–3 .
